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Ung (Samuel et al., 2003b), kidney (Samuel et al., 2004b), and heart (Samuel et al., 2004a) by the administration of relaxin. In the lung, treatment with relaxin reduces expression of collagen types I and III, increases levels of MMPs, and reduces fibrosis (Unemori et al., 1996). In kidney-derived fibroblasts, relaxin inhibits profibrotic changes induced by TGF-b by a mechanism involving the NO/gu

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