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We measured gene expression corresponding to insulin and IGF polypeptides and receptors, and insulin receptor substrates (IRSs) that transmit signals required for growth, survival, energy metabolism, and neuronalELISAs were used to measure sustained effects of NDEA treatment and/or chronic HFD feeding on Tau, phospho-Tau, AbPP, AbPP-Ab, ChAT, and AChE levels in brain tissue. Early limited exposur
S. Nutr Rev 2007, 65(12 Pt 2):S152-156. 2. Launer LJ: Next steps in Alzheimer's disease research: interaction between epidemiology and basic science. Curr Alzheimer Res 2007, 4(2):141-143. 3. Wang XP, Ding HL: Alzheimer's disease: epidemiology, genetics, and beyond. Neurosci Bull 2008, 24(2):105-109. 4. de la Monte SM, Neusner A, Chu J, Lawton M: Epidemilogical Trends Strongly Suggest Exposures a
S insipidus, and optic atrophy in a brother and sister. Am J Dis Child 1977, 131(12):1382-1385. 75. de la Monte SM, Tong M, Cohen AC, Sheedy D, Harper C, Wands JR: Insulin and insulin-like growth factor resistance in alcoholic neurodegeneration. Alcohol Clin Exp Res 2008, 32(9):1630-1644. 76. de la Monte SM: Insulin resistance and Alzheimer's disease. BMB Rep 2009, 42(8):475-481. 77. Giovannone B

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